Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: The effects of CaMKII and angiotensin AT1 receptor inhibition

Tomas Rajtik; Slavka Carnicka; Adrian Szobi; Zoltan Giricz; Jin O-Uchi; Veronika Hassova; Pavel Svec; Peter Ferdinandy; Tanya Ravingerova; Adriana Adameova

doi:10.1016/j.dib.2016.03.017

Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: The effects of CaMKII and angiotensin AT₁ receptor inhibition

Tomas Rajtik, Slavka Carnicka, Adrian Szobi, Zoltan Giricz, Jin O-Uchi, Veronika Hassova, Pavel Svec, Peter Ferdinandy, Tanya Ravingerova, Adriana Adameova

Research output: Contribution to journal › Article › peer-review

3 Scopus citations

Abstract

Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT₁ receptor inhibitor. This data article provides information in support of the original research article "Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT₁ receptor-NOX2 signaling axis" [1].

Original language	English (US)
Pages (from-to)	730-734
Number of pages	5
Journal	Data in Brief
Volume	7
DOIs	https://doi.org/10.1016/j.dib.2016.03.017
State	Published - Jun 1 2016
Externally published	Yes

Bibliographical note

Publisher Copyright:
© 2016 The Authors.

Keywords

AT receptor blockade
Apoptosis
CaMKII inhibition
Myocardial ischemia/reperfusion injury
Necrosis

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10.1016/j.dib.2016.03.017

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Cite this

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title = "Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: The effects of CaMKII and angiotensin AT1 receptor inhibition",

abstract = "Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article {"}Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis{"} [1].",

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doi = "10.1016/j.dib.2016.03.017",

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T1 - Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury

T2 - The effects of CaMKII and angiotensin AT1 receptor inhibition

AU - Rajtik, Tomas

AU - Carnicka, Slavka

AU - Szobi, Adrian

AU - Giricz, Zoltan

AU - O-Uchi, Jin

AU - Hassova, Veronika

AU - Svec, Pavel

AU - Ferdinandy, Peter

AU - Ravingerova, Tanya

AU - Adameova, Adriana

PY - 2016/6/1

Y1 - 2016/6/1

N2 - Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article "Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis" [1].

AB - Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article "Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis" [1].

KW - AT receptor blockade

KW - Apoptosis

KW - CaMKII inhibition

KW - Myocardial ischemia/reperfusion injury

KW - Necrosis

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